All Articles Tagged As: cancer
 | Berkeley Lab researchers have discovered a rotational motion that plays a critical role in the ability of breast cells to form the spherical structures in the mammary gland known as acini. This rotation, called "CAMo," for coherent angular motion, is necessary for the cells to form spheres. Otherwise, cells undergo random motion, leading to loss of structure and malignancy. ...> Full Article |
 | Cancer cells tend to take up more glucose than healthy cells, and researchers are increasingly interested in exploiting this tendency with drugs that target cancer cells' altered metabolism.
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The human T-lymphotropic virus type 1, a cancer-causing retrovirus, exploits key proteins in host cells to extend the life of those cells, thereby prolonging its own survival and ability to spread, according to a new study. The virus, which causes adult T-cell leukemia and lymphoma, produces a protein called p30 that targets two important cell proteins, one involved in DNA damage repair, the other involved in the destruction of proteins within the cell.
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In a new paper published Jan. 21 in the journal Science, a team of researchers led by Microbiology and Immunology professor Blossom Damania, Ph.D., has shown for the first time that the Kaposi sarcoma virus has a decoy protein that impedes a key molecule involved in the human immune response.
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 | The cell signaling pathway known as Wnt, commonly activated in cancers, causes internal membranes within a healthy cell to imprison an enzyme that is vital in degrading proteins, preventing the enzyme from doing its job and affecting the stability of many proteins within the cell, researchers at UCLA's Jonsson Comprehensive Cancer Center have found.
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Scientists recreate the very first step by which a normal cell transforms itself into a cancer cell.
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 | Microbes matter -- perhaps more than anyone realizes -- in basic biological development and, maybe, they could be a target for reducing cancer risks, according to University of Oregon researchers. ...> Full Article |
Scientists have discovered that an enzyme can rid cells of a gene believed to be responsible for a wide range of cancers.
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A study investigating how a cellular enzyme affects blood glucose levels in mice provides clues to pathways that may be involved in processes including the regulation of longevity and the proliferation of tumor cells. In their report in the Jan. 22 issue of Cell, researchers describes the mechanism by which absence of the enzyme SIRT6 induces a fatal drop in blood sugar in mice by triggering a switch between two critical cellular processes.
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